Fig. 1. Images show incisor drifting and gingival recession (A and B) in a 20-year-old Thoroughbred gelding with history of excessive dentistry (incisor and canine reductions). Radiographs of the same horse are shown of the maxillary (C) and mandibular (D) incisors showing resorption and lysis of teeth and bone. Radiographs were taken from a 24-year-old Quarter Horse mare with minimal outward clinical signs (history of avoidance behavior around her mouth). Both maxillary (E) and mandibular (F) incisors exhibit severe hypercementosis of the corner incisors, resulting in crowding of central incisors and loss of rostral incisive and mandibular bone. F, Remnant of canine 304 is superimposed over 303.
the inflammation of the periodontal ligaments were instrumental in the cause of resorptive lesions in calcified incisors.2,9 Studies were performed by Stazyk in 2008, whereby the diseased teeth were analyzed histologically and grossly. The surround- ing cementum was histologically different in af- fected teeth when compared with normal teeth. The type of collagen fibers as well as the arrange- ment and size of blood vessels were different. The arrangement of fibers were disordered, and the vas- cular channels were large within the diseased ce- mentum. An independent variable asked on the
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retrospective study involved the question of whether or not the horse had ever been on pasture. It is possible that the lack of grazing with the associated tug and tearing motion and lack of movement with the incisors may be associated with a stagnant ef- fect, thereby producing a venous congestion with enlarged vascular channels and lack of blood pump- ing into the gums. Our hypothesis is that the pres- ence of periodontal disease is a risk factor that may be a predictor of future EOTRH. The development of periodontal disease may be multifactorial and may not only include lack of blood supply to the
